When I visited the University of Texas, I had the pleasure of chatting with Anthony Lacagnina (from Michael Drew‘s lab). He and his colleagues have been producing the Brain Matters podcast for over a year, and have now interviewed lots of neuroscientists. If you’re interested in hearing about neurogenesis, forgetting, and football (soccer) then listen away (also here on itunes, episode 23).
The Josselyn/Frankland lab celebrated Festivus in style yesterday at the traditional location (The Red Room on Spadina). Handed the festivus pole, Colleen launched into a long list of grievances in spectacular fashion, before taking on all-comers in the feats of strength. More pictures are posted here.
John Howland has spent the last 4 months in the lab as
Which neurons become part of an engram or memory trace? Is this process random? Or are neurons ‘pre-selected’? In this short review published in Neuropsychopharmacology, we discuss the rules of allocation. A pdf version is available here.
Our new paper published last week in the Journal of Neuroscience. Below: Clarified olfactory bulbs (J. Epp).
Autism Spectrum Disorders (ASDs) are thought to be caused by the abnormal development or dysfunction of synapses. Consistent with these many genes that code for key synaptic proteins confer increased risk for ASD. Adding to story, a new study shows that deleting an ASD-risk gene called Sema5A causes neurons to form more connections in mice, and also alters how these mutant mice interact with one another. Justin Kenney wrote a commentary on this paper, and it is published in eLife.
From top left: 1) Steve Ramirez and Karim Nader; 2) Kiriana Cowansage and Josh Johansen; 3) Jonathan Britt and Kay Tye; 4) Jaideep Bains and Shernaz Bamji; 5) Natalie Tronson and Satoshi Kida; 6) Megan and Hayley from the Matt Hill lab.