The Josselyn/Frankland lab celebrated Festivus in style yesterday at the traditional location (The Red Room on Spadina). Handed the festivus pole, Colleen launched into a long list of grievances in spectacular fashion, before taking on all-comers in the feats of strength. More pictures are posted here.
John Howland has spent the last 4 months in the lab as
hipster-in-residence a visiting scientist during his sabbatical from the University of Saskatchewan. It’s been a complete pleasure having John in the lab, and we are sad to see you go!
Which neurons become part of an engram or memory trace? Is this process random? Or are neurons ‘pre-selected’? In this short review published in Neuropsychopharmacology, we discuss the rules of allocation. A pdf version is available here.
Lina took to the whiteboard yesterday…
Our new paper published last week in the Journal of Neuroscience. Below: Clarified olfactory bulbs (J. Epp).
Autism Spectrum Disorders (ASDs) are thought to be caused by the abnormal development or dysfunction of synapses. Consistent with these many genes that code for key synaptic proteins confer increased risk for ASD. Adding to story, a new study shows that deleting an ASD-risk gene called Sema5A causes neurons to form more connections in mice, and also alters how these mutant mice interact with one another. Justin Kenney wrote a commentary on this paper, and it is published in eLife.
Clockwise from top left: Axel, Chen, Liz, Leo and Asim.
From top left: 1) Steve Ramirez and Karim Nader; 2) Kiriana Cowansage and Josh Johansen; 3) Jonathan Britt and Kay Tye; 4) Jaideep Bains and Shernaz Bamji; 5) Natalie Tronson and Satoshi Kida; 6) Megan and Hayley from the Matt Hill lab.
Jonathan fielding questions at his posters in 2013 and 2014. Left, 2013 without Karl Deisseroth as co-author. Right, 2014 with Karl Deisseroth as co-author.